4 edition of Molecular mechanisms of metabolic arrest found in the catalog.
Molecular mechanisms of metabolic arrest
Includes bibliographical references and index.
|Statement||edited by K.B. Storey.|
|Series||Experimental biology reviews|
|Contributions||Storey, K. B.|
|The Physical Object|
|Pagination||xiii, 199 p. :|
|Number of Pages||199|
Conference abstracts, book chapters, and unpublished results were not included. Only English language articles were included in this review. Major keywords used in various combinations included: genistein, cancer, anticancer, molecular mechanisms prevention, treatment, in vivo, in vitro, and clinical studies. The bibliography of the primary Cited by: 1. This thesis aims to highlight molecular mechanisms that have been altered by prenatal undernutrition and may be involved in the metabolic syndrome. Two sepa- rate studies were conducted both using a rat model developed through manipulation of the maternal diet to provoke the key features of the metabolic syndrome in adult o spring. Research Area A: Mechanisms of tumor development: intrinsic and extrinsic control of cell proliferation and tissue invasion; Research Area B: Principles of immunity, inflammation and infection; Research Area C: Molecular mechanisms and metabolic control of tissue degeneration and regeneration. Miguel A Alejandre Alcazar - CAP Title:From Natural Products to Designer Drugs: Development and Molecular Mechanisms Action of Novel Anti-Microtubule Breast Cancer Therapeutics VOLUME: 17 ISSUE: 22 Author(s):Tejashree Mahaddalkar and Manu Lopus* Affiliation:Experimental Cancer Therapeutics and Chemical Biology, UM-DAE Centre for Excellence in Basic Sciences, University of Mumbai Kalina Campus, Mumbai, Experimental Cancer Cited by: 6.
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Molecular Mechanisms of Metabolic Arrest focuses on the latest opinions of molecular mechanisms and principles of dormancy regulation, highlighting various new concepts in metabolic rate control.
The mechanisms used by these organisms to decrease metabolic rates and to survive at these lower rates is a fascinating biological question. The book Molecular mechanisms of metabolic arrest: life in limbo, edited by K. Storey, attempts to tackle this : Bernard B.
Rees. Buy Molecular Mechanisms of Metabolic Arrest: Life in Limbo (Society for Experimental Biology) on FREE SHIPPING on qualified orders. Molecular Mechanisms of Metabolic Arrest focuses on the latest opinions of molecular mechanisms and principles of dormancy regulation, highlighting various new concepts in metabolic rate control.
This book will strongly appeal to all molecular and comparative scientists studying organismal responses to environmental stress. Molecular Mechanisms of Metabolic Arrest focuses on the latest opinions of molecular mechanisms and Many organisms can reduce their resting metabolic rate to a state of suspended animation to allow them to survive in adverse environmental conditions.
Many organisms can reduce their resting metabolic rate to a state of suspended animation to allow them to survive in adverse environmental conditions. Molecular Mechanisms of Metabolic Arrest focuses on the latest opinions of molecular mechanisms and principles of dormancy regulation, highlighting v.
Recommended Citation. Rees, B.B. Molecular mechanisms of metabolic arrest: life in limbo (book review). Journal of Experimental Biology Author: Bernard Rees.
Liver Regeneration and Carcinogenesis Molecular and Cellular Mechanisms. the cells are highly differentiated and in a state of growth arrest. They are induced to enter the cell cycle by cell loss or functional inadequacy, for example because of surgical resection, infections, toxic or physical injury, or to metabolic imbalances caused by.
Apoptosis: Molecular Mechanisms. Chapter contrast, apoptosis is an active, metabolic, genetically. alternatively cell cycle arrest or apoptosis (Figure 3). In cell. Molecular Mechanisms of Metabolic Arrest focuses on molecular mechanisms and principles of dormancy regulation, highlighting various concepts in metabolic rate control.
This book will strongly appeal to all molecular and comparative scientists studying organismal responses to environmental stress. Molecular mechanisms of metabolic regulation by insulin in Drosophila. Teleman AA(1). Author information: (1)German Cancer Research Center (Deutsches Krebsforschungszentrum), Im Neuenheimer FeldHeidelberg, Germany.
[email protected] The insulin signalling pathway is highly conserved from mammals to by: Neuroblastoma: Molecular Mechanisms and Therapeutic Interventions comprehensively reviews current concepts in molecular and histopathological mechanisms that influence the growth of human malignant neuroblastoma, along with exciting therapeutic interventions.
This book features a broad collection of contributions from leading investigators in. Apoptosis, derived from the Greek word for the natural process of leaves falling from trees, Molecular mechanisms of metabolic arrest book a distinct form of programmed cell death (Kerr et al., ; Kerr, ).Although such programmed deaths were described many decades ago, the significance of apoptosis was largely overlooked, in particular, its relevance to disease.
Molecular Mechanisms of Metabolic Arrest in Molluscs (K.B. Storey). Inhibition of Protein Synthesis in Isolated Hepatocytes as an Immediate Response to Oxygen Limitation (P. Buc-Calderon, V. Lefebvre, and M. van Steenbrugge). Control of Metabolic Rate by Multienzyme Complexes: Is Glycolysis in Hypoxia and Anoxia Regulated by Complex Formation.
Conference abstracts, book Molecular mechanisms of metabolic arrest book, and unpublished results were not included. Only English language articles were included in this review. Major keywords used in various combinations included: genistein, cancer, anticancer, molecular mechanisms prevention, treatment, in vivo, in vitro, and clinical studies.
The bibliography of the primary Cited by: 1. Cellular senescence is the dynamic process of durable cell-cycle arrest. Senescent cells remain metabolically active and often acquire a distinctive bioactive secretory phenotype. Much of our molecular understanding in senescent cell biology comes from studies using mammalian cell lines exposed to stress or extended culture periods.
Kenneth B. Storey, Ph.D., FRSC, Canada Research Chair in Molecular Physiology (Tier I), is a Canadian scientist whose work draws from a variety of fields including biochemistry and molecular biology.
Storey has a world-wide reputation for his research on biochemical adaptation - the molecular mechanisms that allow animals to adapt to and endure severe environmental stresses such as deep cold. By admin MEDICAL BOOKS 0 Comments Regulatory Toxicology, Third Edition This practical book provides toxicologists with essential information on the regulations that govern their jobs and products.
Regulatory Toxicology, Third Edition is an up-to-date guide to required safety assessment for the entire range of man-made marketed products. The precise molecular mechanisms of how MYC induces apoptosis remain unclear.
However, it appears that multiple pathways are regulated by MYC, including one requiring the p53 tumour suppressor, where MYC‐induced apoptosis is preceded by stabilization of p In mammalian cells, p53 is a major regulator of cell cycle arrest and apoptosis (see Cited by: 3.
Metabolic arrest is achievable by means of a reversed or negative Pasteur Effect (reduced or unchanging glycolytic flux at reduced O2 availability) while coupling of metabolic and membrane function is achievable in spite of the lower energy turnover rates by maintaining membranes of low permeability (probably via reduced densities of ion.
The Example of Brown Adipose Tissue / A. Malan --Ch Environmental and Exercise Anaerobiosis in Frogs / G. Wegener / U. Krause --Ch Role of Metabolic Acidosis in the Buffering of ATP by Phosphagen Stores in Fish: An In Vivo NMR Study / A.
van Waarde / G. van den Thillart --Ch Molecular Mechanisms of Metabolic Arrest in Mollusks / K.B. Bottom Line: Such strategies include reversible post-translational modifications as well as the implementation of translation and transcription controls facilitating metabolic uentially, the molecular mechanisms controlling these processes have profound clinical review article will discuss the theory of cell cycle arrest in anoxic vertebrates and more.
But aestivation is much more than remaining inactive. Successful aestivation requires the selection of a proper microhabitat, variable degrees of metabolic arrest and responsiveness to external stimuli, the ability to sense the proper time of year for emergence, the preservation of.
Polycystic ovary syndrome (PCOS) is a common endocrinopathy of unknown aetiology that affects women of reproductive age. During the past ten years, defective insulin activity in PCOS has been demonstrated in target tissues and causes insulin resistance and hyperinsulinaemia.
Furthermore, presence of insulin receptors in the ovarian tissue and overproduction of androgens by theca cells Cited by: For the last few years, a large number of studies have deepened and progressed our knowledge of the molecular mechanisms involved in the development of metabolic disorders.
In this regard, special attention has been focused on oxidative stress, inflammation, endoplasmic reticulum stress, alterations on glucose and/or lipid metabolism, insulin.
In this chapter, we discuss the molecular mechanisms of angiogenesis in glioblastoma and recently developed approaches inducing an antiangiogenic response such as protein tyrosine kinase.
The third edition of The Molecular Biology of Cancer: Mechanisms, Targets, and Therapeutics offers a fresh approach to the study of the molecular basis of cancer, by showing how our understanding of the defective mechanisms that drive cancer is leading to the development of.
Overview. Professor Donald Jackson received his B.S. in Biology at Geneva college in Following the receipt of his Ph.D. in Physiology in from the University of Pennsylvania, he spent two years at Duke University as a Research Associate in Zoology.
Animals have to cope with starvation. The molecular mechanisms by which animals survive long-term starvation, however, are not clearly understood. When they hatch without food, C. elegans arrests development at the first larval stage (L1) and survives more than two weeks.
Here we show that the survival span of arrested L1s, which we call L1 longevity, is a starvation response regulated by Cited by: Moreover, molecular mechanisms causing these metabolic changes are just starting to be unraveled. This chapter presents recent findings on molecular markers that have been involved in the mechanisms of proliferation and energy metabolism of oral cancer and in addition provides new perspectives on oral cancer diagnosis and : Masakatsu Fukuda, Hideaki Sakashita.
Kenneth B. Storey, Ph.D. FRSC (born Octo ) is a Canadian scientist whose work draws from a variety of fields including biochemistry and molecular is a Professor of Biology, Biochemistry and Chemistry at Carleton University in Ottawa, Canada. Storey has a world-wide reputation for his research on biochemical adaptation - the molecular mechanisms that allow animals to adapt Born: Kenneth Bruce Storey, Octo (age.
Third, like other cancers, HCC undergoes a dynamic process changing morphology and molecular features as it advances. Therefore, molecular mechanisms of hepatocellular carcinogenesis may vary depending on different factors and this is probably why a large set of mechanisms have been associated with these by: 3.
Book: Cells - Molecules and Mechanisms (Wong) Cell Cycle Expand/collapse global location most of the metabolic activity of the cell is shut down, and the cell concentrates its resources on dividing the nuclear and cellular material equally to support the life of both resulting daughter cells.
The oocytes arrest for about 8 months. Asparaginase is an integral part of the treatment for acute lymphoblastic leukemia, the most common childhood cancer. Asparaginase produces hepatotoxicity resulting in treatment-related metabolic complications that include fatty liver, reduced plasma proteins, and coagulation problems that lead to thromboembolism and cerebrovascular events.
Introduction. Congenital heart disease (CHD) is the single most common class of birth defects and one of the leading causes of infant mortality. 1,2 The majority of CHDs involve lesions in which the loading conditions on the ventricles are abnormal.
Despite major advances in surgical techniques, the repair of these lesions is often imperfect, leading to lifelong chronic pressure and volume Cited by: In this review, we summarize the molecular mechanisms of breast cancer organotropic metastasis by focusing on tumor cell molecular alterations, Cited by: Background: Modern lifestyles, especially high-caloric intake and physical inactivity, contribute to the increased prevalence of non-alcoholic fatty liver disease (NAFLD), which becomes a significant health problem worldwide.
Lifestyle changes, however, affect not only parental generation, but also their offspring, reinforcing the need for. It will be essential reading for researchers and students in biochemistry, molecular biology, microbiology, cell biology, nutrition and medical sciences. Other interested groups include biological inorganic chemists with an interest in iron metabolism, health professionals with an interest in diseases of iron metabolism, or of diseases in which Manufacturer: Wiley.
These mechanisms include its inhibitory effects on the insulin growth factor-1 (IGF-1), G protein-coupled receptor (GPCR) and mTORC1 signaling pathways .
We then discuss the metabolic effects of metformin in cancer. For example, metformin has been shown Cited by: 2. Surviving Hypoxia: Mechanisms of Control and Adaptation is a synthesis of findings and thoughts concerning hypoxia. The thermodynamics of hypoxia are discussed in detail, including acid-base balance and self-pollution resulting from the accumulation of anaerobic end-products.
The book focuses on des. Molecular characterization of signaling pathways regulating dauer arrest Genetic analysis of dauer-constitutive (Daf-c) and dauer-defective (Daf-d) mutants has defined four distinct pathways that regulate dauer arrest (Gerisch et al., ; Gottlieb and Ruvkun, ; Jia et al., ; Riddle et al.,File Size: 1MB.Aim.
The Clinical, Metabolic and Molecular Physiology (CMMP) research group aims to determine the mechanisms regulating mass and function of the musculoskeletal system, with a particular focus on age and age-associated conditions.
Specifically, the group endeavours to uncover the mechanisms regulating alterations in protein and energy metabolism with ageing and disease by combining.Title:Molecular Mechanisms Involved in the Aging of the T-cell Immune Response VOLUME: 13 ISSUE: 8 Author(s):Marco Antonio Moro-Garcia, Rebeca Alonso-Arias and Carlos Lopez-Larrea Affiliation:Immunology Department, Hospital Universitario Central de Asturias, Oviedo, Spain.
Keywords:Differentiation, Epigenetic regulation, Immunosenescence, T-cell signalling, Telomere .